Figure 2.
Changes in the protein levels of GRP78, CHOP, and the conversion of LC3‐I to LC3‐II in perihematoma tissues in the rat intracerebral hemorrhage (ICH) model. (A) GRP78 and CHOP protein levels in perihematoma tissues of the rat ICH model determined by Western blots. (B, C) Results of quantitative analysis of (A) Bars represent mean±SEM, *P<.05, **P<.01 compared with the sham group; #P<.05, n=6. (D) The protein level of LC3 in perihematoma tissues of the rat ICH model determined by Western blots. (E) Quantitative analysis of the conversion of LC3‐I to LC3‐II shown in (D) Bars represent mean±SEM, *P<.05, **F 8,48=28.13, P<.01 compared with the sham group; ##F 8,48=20.02, P<.01, n=6. (F) Western blot assay of the protein level of LC3 in perihematoma tissues of rats with indicated treatment. (G) Results of quantitative analysis of F. Bars represent mean±SEM, *P<.05, **F 7,40=29.53, P<.01 compared with the sham group; #F 7,40=11.12, P<.05, ##F 7,40=27.25, P<.001, &F 7,40=12.71, P<.05, N.S.=No significant differences, n=6. CHOP, C/EBP homologous protein; GRP78, glucose‐regulated protein 78; LC3, microtubule‐associated protein light‐chain 3; Nor, normal