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. Author manuscript; available in PMC: 2016 Sep 1.
Published in final edited form as: Exp Eye Res. 2015 Jul 4;138:104–113. doi: 10.1016/j.exer.2015.07.001

Figure 1. Suppression of cloche lens phenotypes by expression of Rno.Cryaa transgene.

Figure 1

Cloche mutants showed apparent lens defects (A) and transgenic expression of Tg(cryaa:Rno.Cryaa) rescued, at least partially, the lens defects in cloche mutants (B, E). Zoomed-in images (C, D) of the lenses in cloche−/− and cloche−/−; Tg(cryaa:Rno.Cryaa), denoted by white-dotted boxes (A, B). Embryos of lens-specific Rno.Cryaa transgene displayed Cerulean marker in the heart (F; Tg(cryaa:Rno.Cryaa,myl7:Cerulean), white arrow)

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