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. Author manuscript; available in PMC: 2013 May 19.
Published in final edited form as: Cell Host Microbe. 2008 Apr 17;3(4):224–232. doi: 10.1016/j.chom.2008.03.003

Fig. 5. M. bovis BCG inhibits inflammasome agonist-dependent activation of caspase-1.

Fig. 5

A. RAW264.7 cells were treated with inflammasome agonists nigericin (20 μM) or ATP (1μM), infected with M. bovis BCG for 4h where indicated, and caspase-1 activation assessed by immunoblotting. M. bovis BCG: zmp1+ (wild type, untreated); zmp1++Cm, wild type attenuated by overnight tretment with 30 μg/ml chloramphenicol. B. Densitometric ratio of active caspase-1 (p20) to β-actin.

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